Talk:Trypanosoma cruzi

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Needs more info[edit]

Someone needs to add information about the relationships to Achalasia and Hirschprung's disease. A discussion of T. Cruzi toxin is also warranted.

Source: I am a medical doctor and researcher. — Preceding unsigned comment added by 71.221.247.183 (talk) 17:39, 21 May 2012 (UTC)[reply]

Copyvio[edit]

The copyvio looks pretty blatant, but the article before that point was fine. Do we really need to wait while the copyvio is considered before reverting to the previous known good version? If permission to GFDL the text is later given it can easily be added. - Taxman ^Talk 23:18, 28 March 2006 (UTC)[reply]

I sorta doubt permission will be given. See their copyright statement [1]. The material was copied into WP by User:Kloe who has only 4 edits, all on 15 Jan 2006. What a waste. Mccready 16:13, 2 April 2006 (UTC)[reply]

1.[Carlos Chaga]s cardiopathy appears to be an expedient example of selective [autonomic] insufficiency of the myocardium. 2.Myocardial autonomic insufficiency is subject to a wide array of pathophysiology. 3.Posit that measured myocardial autonomic performance can be further divided into an internal, sinoatrially triggered forward, [sympathetic] polarization pathway [systole] and an external, backward, [parasympathetic] repolarization pathway fielding and returning brainstem influence initiated through the thoracic spinal accessory nerves and vagus nerve[diastole].4.Chagas/T. Cruzi appears to represent an especially virulent and complex parasitic entity specifically targeted towards [histology cardiac] autonomic nerves. 5.Incredible geometrical remodeling of the heart, esophagus and colon are manifest as hallmarks of incremental loss of autonomic control of these organs under this chiefly parasympathetic pathophysiologic burden.6. Background cardiac innervation and supply from the spinal accessories and vagal input (brainstem origin) as an opposed electrical entity of countercontrol of the myocardium is posited as a more complete literal rendering of [diastole]. 7.Chagas represents slow, incremental degradation and decline of parasympathetic countercontrol [diastole] of the myocardium. 8.Utility of study of T. Cruzi is posited as a model of diastolic, nonischemic heart failure. 9.Posited proof as an {almost mathematically pure} model of diastolic dysfunction by Chagas is exhibited by the {early} complete sparing of the internal sinoatrial/atrioventricular/sympathetic-systolic pathways. 10. Chagas is an expedient model of accelerated diastolic failure as readily demonstrated by ease of invasion [virulence] upon vagal influence to the individual branches of the opposed autonomic electrical trees at work in the myocardium. 11.[Histologic] work spanning the length of the vagus illustrates the predilection of this parasite for very specialized nervous tissue. 12. Autonomic insufficiency inevitably leads to negative geometric remodeling. 13. Chagas myocardial disease is perhaps the most compelling of the [nonischemic cardiomyopathy]s solely based on the numbers of afflicted South Americans. Research in this area is promising.--Lbeben (talk) 01:33, 28 April 2008 (UTC)--Lbeben (talk) 01:36, 28 April 2008 (UTC)[reply]

Geographical distribution?[edit]

I learned recently that Chagas Disease, common in S. America is now being seen further north. I guess that means this organism might now be making its way north too. Where has its natural habitat around the globe been until now? -thanks, Onceler 20:27, 24 October 2007 (UTC)[reply]

I work a few days per month in a large community care office in South Carolina. We see a significant immigrant population and are beginning to see Chagasic heart failure and gastrointestinal disease migrate north as a result. US evidence based medicine seems a bit thin on this topic. Where can I find common sense guidelines regarding medicine in addressing these patients?--Lbeben (talk) 02:11, 11 January 2008 (UTC)[reply]

Blacklisted Links Found on the Main Page[edit]

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